Endocannabinoids

Introduction

Endocannabinoids are naturally occurring compounds within the human body that play a crucial role in maintaining internal stability and health by interacting with the endocannabinoid system (ECS). Two of the most researched and significant endocannabinoids are Anandamide (AEA) and 2-Arachidonoylglycerol (2-AG). These molecules are pivotal in various physiological processes, from memory and mood regulation to pain and inflammation management. 

Anandamide (AEA)

Anandamide was first identified and named by the eminent scientist Raphael Mechoulam. The name is derived from the Sanskrit word "Ananda," which means "joy, delight, or bliss," reflecting its impact on mood and well-being. Interestingly, it is produced in the human body and found in small quantities in chocolate.

Anandamide is synthesized in cell membranes from arachidonic acid. It is metabolized primarily by the enzyme Fatty Acid Amide Hydrolase (FAAH), which breaks it down and regulates its levels and effects in the body.

Anandamide acts as an agonist at CB1 cannabinoid receptors, located primarily in the brain and throughout the central nervous system, and at TRPV1 receptors, which regulate pain, body temperature, and inflammation.

Anandamide influences several critical physiological processes:

2-Arachidonoylglycerol (2-AG)

Mechoulam's group also discovered 2-arachidonoylglycerol. They noted its ability to bind to both CB1 and CB2 receptors, suggesting its broad role in modulating the central nervous and immune systems. 2-AG is present at significantly higher brain concentrations than anandamide—about 170 times higher. Like anandamide, it is synthesized on demand from arachidonic acid in cell membranes.

2-AG is primarily broken down by Monoacylglycerol Lipase (MAGL), an enzyme that regulates its levels and activity within the body.

2-AG is a potent activator of both CB1 and CB2 receptors. Its activity at CB2 receptors influences a broader range of physiological functions than anandamide, mainly in the peripheral organs and cells associated with the immune system.

2-Arachidonoylglycerol influences critical physiological processes:

Endocannabinoid-Like

Additionally, the metabolism of acetaminophen, a common pain reliever, produces a metabolite known as N-arachidonoylaminophenol. This derivative acts like an endocannabinoid, inhibiting COX enzymes and activating TRPV1, further illustrating the interconnections between pharmacology and endocannabinoid pathways.